- What are the different ways to account for SES in an analytic model when investigating racial/ethnic health disparities? (Hint: you should have three options). Discuss the interpretations/implications of each approach as it relates to the interest in understanding health disparities by race/ethnicity.
SES may act as confounder or mediator, It could also consider the main outcome of measure. For example in the Headen et al paper, SES acts as a confounder, because correlates directly with both the dependent variable (pregnancy weight gain) and an independent variable (racial/ethnic disparities) in the study. The mediator effect is seen in Lorch et al paper, because the influence of racial and ethnic disparities in fetal death is mediated by SES meaning that the observed relationship between the dependent variable (pregnancy weight gain) and an independent variable (racial/ethnic disparities) is via the inclusion of SES, so it is not a direct causal relationship between the independent variable and the dependent variable. MLRA, as seen in Merlo et al, could be helpful to understand the influence of SES on the outcome variable.
- Think about multilevel influences on a health outcome of interest to you. Discuss how you would study this, including measurement and analytic approaches you would use to account for exposures across multiple levels.
In relation to my research area, I will be interested in the differences in long-term survival in patients with dementia according to SES. I think that patients with short-term survival after diagnosis will have low SES, anyway this will be a complex analysis so a MLRA analysis will be necessary to control for access to health care, community, and social support, educational differences, progression rate etc.