epigenetic regulation, allostatic load, GxE

epigenetic regulation, allostatic load, GxE

by Hala Borno -
Number of replies: 1

1.     Weaver et al propose that among rats, maternal behavior towards newborn pups influences their cortisol response to stress via epigenetic mechanisms that change the expression of glucocorticoid receptor gene for the rest of the pup’s life.  They argue that because epigenetic patterns are established at specific developmental periods, there is extreme time sensitivity to when the pup is exposed to particular maternal behaviors (licking and grooming, in this case), and maternal behavior before or after that sensitive period window is not as important.   Do you think this mechanism is relevant in humans?  If so, what behaviors are most analogous to “maternal licking and grooming”? 

I do think this mechanism is relevant to humans and that epigenetic patterns may be established at specific development periods. “Maternal licking and grooming” are examples of physical care and affection from rat to pup. Analogous human behavior are hugs, patting on the back, and kisses.

2.     Gruenewald, in contrast, emphasize the cumulative effects of SES adversity on a multi-system allostatic load measure.   Do you think that the Gruenewald findings are consistent, inconsistent, or unrelated to the Weaver findings?  Explain. 

Gruenewald’s findings support the cumulative risk hypothesis, that greater experience of SES adversity across the life course may cumulate to have a greater negative effect on biological functioning in later adulthood.  I think that Weaver and Gruenewalds findings complement one another. We know that epigenetic mechanisms are dynamic and therefore it is possible that cumulative explore of SES adversity may change epigenetics and therefore protein expression at different life points.

3.     Hertzmann and Boyce argue that “it is not genes or environment, nor is it genes and environment, but rather it is gene-by-environment interactions that influence developmental trajectories.”  To what extent do you think that GxE interactions can contribute to major disparities along racial/ethnic, socioeconomic, or geographic dimensions?

In short, I think GxE interactions influence disparities observed in all dimensions. I believe that contexts, whether socially constructed (race/ethnicity), socioeconomic, or geographic are subject to epigenetic regulation simply due to the fact that these dimensions are a reflection of environment. Hence GxE is a major driver of disparities.


In reply to Hala Borno

Re: epigenetic regulation, allostatic load, GxE

by Maria Glymour -

Thanks Hala.  The cumulative effects model, if true, is very important because it suggests we can still substantially offset early life harm via adult interventions.  The critical period model suggests once the window is closed, there is little to be done.  This debate has informed much recent emphasis on early childhood interventions - because a major argument from Heckman and others has been that adult social interventions appear to have little to no effect, in contrast to interventions ages 3-5.  Epigenetic patterns do change in adulthood but much much less than in early life.  Epigenetic erasure/rewriting is highly regulated biologically with respect to developmental timing, so it's possible that action really has to be in early life and everything later is unlikely to have large effects. 


Maria